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function-blocking antibodies targeting tlr2 tl2.1  (Thermo Fisher)


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    Thermo Fisher function-blocking antibodies targeting tlr2 tl2.1
    Mechanisms of LPS-induced A549 proliferation. A549 cells were either sham-incubated ( control ) or exposed to 10 μg/ml of LPS ( n = 3 for LPS 0111:B4 and n = 3 for LPS F515, total n = 6) in the absence or presence of neutralizing antibodies targeting <t>TLR2,</t> TLR4, CD14 or EGFR. After 24 h of incubation proliferation was quantified by determining MTS activity. All data are expressed as percentage of unstimulated cells ( control ). Mean ± SEM of six independent experiments are given
    Function Blocking Antibodies Targeting Tlr2 Tl2.1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/function-blocking antibodies targeting tlr2 tl2.1/product/Thermo Fisher
    Average 90 stars, based on 1 article reviews
    function-blocking antibodies targeting tlr2 tl2.1 - by Bioz Stars, 2026-03
    90/100 stars

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    1) Product Images from "Endotoxin induces proliferation of NSCLC in vitro and in vivo: role of COX-2 and EGFR activation"

    Article Title: Endotoxin induces proliferation of NSCLC in vitro and in vivo: role of COX-2 and EGFR activation

    Journal: Cancer Immunology, Immunotherapy

    doi: 10.1007/s00262-012-1341-2

    Mechanisms of LPS-induced A549 proliferation. A549 cells were either sham-incubated ( control ) or exposed to 10 μg/ml of LPS ( n = 3 for LPS 0111:B4 and n = 3 for LPS F515, total n = 6) in the absence or presence of neutralizing antibodies targeting TLR2, TLR4, CD14 or EGFR. After 24 h of incubation proliferation was quantified by determining MTS activity. All data are expressed as percentage of unstimulated cells ( control ). Mean ± SEM of six independent experiments are given
    Figure Legend Snippet: Mechanisms of LPS-induced A549 proliferation. A549 cells were either sham-incubated ( control ) or exposed to 10 μg/ml of LPS ( n = 3 for LPS 0111:B4 and n = 3 for LPS F515, total n = 6) in the absence or presence of neutralizing antibodies targeting TLR2, TLR4, CD14 or EGFR. After 24 h of incubation proliferation was quantified by determining MTS activity. All data are expressed as percentage of unstimulated cells ( control ). Mean ± SEM of six independent experiments are given

    Techniques Used: Incubation, Control, Activity Assay

    Mechanisms of LPS-induced PGE 2 synthesis. A549 cells were either sham-incubated ( control ) or exposed to 10 μg/ml of LPS ( n = 3 for LPS 0111:B4 and n = 3 for LPS F515, total n = 6) in the absence or presence of neutralizing antibodies targeting TLR2, TLR4, CD14 and EGFR, or the COX inhibitor indomethacin ( indo ) and the specific COX-2 inhibitor NS-398 for 24 h. 8 h before the end of the incubation period, and AA was added. PGE 2 release into the cell supernatant is given in pg/ml. Data are expressed as mean ± SEM of at least six independent experiments
    Figure Legend Snippet: Mechanisms of LPS-induced PGE 2 synthesis. A549 cells were either sham-incubated ( control ) or exposed to 10 μg/ml of LPS ( n = 3 for LPS 0111:B4 and n = 3 for LPS F515, total n = 6) in the absence or presence of neutralizing antibodies targeting TLR2, TLR4, CD14 and EGFR, or the COX inhibitor indomethacin ( indo ) and the specific COX-2 inhibitor NS-398 for 24 h. 8 h before the end of the incubation period, and AA was added. PGE 2 release into the cell supernatant is given in pg/ml. Data are expressed as mean ± SEM of at least six independent experiments

    Techniques Used: Incubation, Control



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    function-blocking antibodies targeting tlr2 tl2.1  (Thermo Fisher)
    90
    Thermo Fisher function-blocking antibodies targeting tlr2 tl2.1
    Mechanisms of LPS-induced A549 proliferation. A549 cells were either sham-incubated ( control ) or exposed to 10 μg/ml of LPS ( n = 3 for LPS 0111:B4 and n = 3 for LPS F515, total n = 6) in the absence or presence of neutralizing antibodies targeting <t>TLR2,</t> TLR4, CD14 or EGFR. After 24 h of incubation proliferation was quantified by determining MTS activity. All data are expressed as percentage of unstimulated cells ( control ). Mean ± SEM of six independent experiments are given
    Function Blocking Antibodies Targeting Tlr2 Tl2.1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/function-blocking antibodies targeting tlr2 tl2.1/product/Thermo Fisher
    Average 90 stars, based on 1 article reviews
    function-blocking antibodies targeting tlr2 tl2.1 - by Bioz Stars, 2026-03
    90/100 stars
    		  Buy from Supplier

    Image Search Results


    Mechanisms of LPS-induced A549 proliferation. A549 cells were either sham-incubated ( control ) or exposed to 10 μg/ml of LPS ( n = 3 for LPS 0111:B4 and n = 3 for LPS F515, total n = 6) in the absence or presence of neutralizing antibodies targeting TLR2, TLR4, CD14 or EGFR. After 24 h of incubation proliferation was quantified by determining MTS activity. All data are expressed as percentage of unstimulated cells ( control ). Mean ± SEM of six independent experiments are given

    Journal: Cancer Immunology, Immunotherapy

    Article Title: Endotoxin induces proliferation of NSCLC in vitro and in vivo: role of COX-2 and EGFR activation

    doi: 10.1007/s00262-012-1341-2

    Figure Lengend Snippet: Mechanisms of LPS-induced A549 proliferation. A549 cells were either sham-incubated ( control ) or exposed to 10 μg/ml of LPS ( n = 3 for LPS 0111:B4 and n = 3 for LPS F515, total n = 6) in the absence or presence of neutralizing antibodies targeting TLR2, TLR4, CD14 or EGFR. After 24 h of incubation proliferation was quantified by determining MTS activity. All data are expressed as percentage of unstimulated cells ( control ). Mean ± SEM of six independent experiments are given

    Article Snippet: In an additional series of experiments in Fig. , function-blocking antibodies targeting TLR2 (clone TL2.1, e-Bioscience, San Diego, CA, USA), TLR4 (clone HTA 125, e-Bioscience, San Diego, CA, USA), CD14 (MY-4, Coulter Immunotech, Hamburg, Germany), EGFR (Cetuximab, Merck Serono, Germany) or COX inhibitors (indomethacin, Sigma, Deisenhofen, Germany and NS-398, Calbiochem, La Jolla, CA, USA) were applied simultaneously to LPS.

    Techniques: Incubation, Control, Activity Assay

    Mechanisms of LPS-induced PGE 2 synthesis. A549 cells were either sham-incubated ( control ) or exposed to 10 μg/ml of LPS ( n = 3 for LPS 0111:B4 and n = 3 for LPS F515, total n = 6) in the absence or presence of neutralizing antibodies targeting TLR2, TLR4, CD14 and EGFR, or the COX inhibitor indomethacin ( indo ) and the specific COX-2 inhibitor NS-398 for 24 h. 8 h before the end of the incubation period, and AA was added. PGE 2 release into the cell supernatant is given in pg/ml. Data are expressed as mean ± SEM of at least six independent experiments

    Journal: Cancer Immunology, Immunotherapy

    Article Title: Endotoxin induces proliferation of NSCLC in vitro and in vivo: role of COX-2 and EGFR activation

    doi: 10.1007/s00262-012-1341-2

    Figure Lengend Snippet: Mechanisms of LPS-induced PGE 2 synthesis. A549 cells were either sham-incubated ( control ) or exposed to 10 μg/ml of LPS ( n = 3 for LPS 0111:B4 and n = 3 for LPS F515, total n = 6) in the absence or presence of neutralizing antibodies targeting TLR2, TLR4, CD14 and EGFR, or the COX inhibitor indomethacin ( indo ) and the specific COX-2 inhibitor NS-398 for 24 h. 8 h before the end of the incubation period, and AA was added. PGE 2 release into the cell supernatant is given in pg/ml. Data are expressed as mean ± SEM of at least six independent experiments

    Article Snippet: In an additional series of experiments in Fig. , function-blocking antibodies targeting TLR2 (clone TL2.1, e-Bioscience, San Diego, CA, USA), TLR4 (clone HTA 125, e-Bioscience, San Diego, CA, USA), CD14 (MY-4, Coulter Immunotech, Hamburg, Germany), EGFR (Cetuximab, Merck Serono, Germany) or COX inhibitors (indomethacin, Sigma, Deisenhofen, Germany and NS-398, Calbiochem, La Jolla, CA, USA) were applied simultaneously to LPS.

    Techniques: Incubation, Control